Blood

Vol. 145, Issue 12 Issue 12 2025
15
Articles

Articles in this Issue

MRD-guided zanubrutinib, venetoclax, and obinutuzumab in relapsed CLL: primary end point analysis from the CLL2-BZAG trial

Moritz Fürstenau, Sandra Robrecht, Christof Schneider et al. Mar 20, 2025 pp. 1282-1292 10.1182/blood.2024026685

Abstract The phase 2 CLL2-BZAG trial tested a measurable residual disease (MRD)–guided combination treatment of zanubrutinib, venetoclax, and obinutuzumab after an optional bendamust...

Math models expose myeloid bias mechanisms in hematopoiesis

Artur C. Fassoni, Ingmar Glauche Mar 20, 2025 pp. 1231-1232 10.1182/blood.2024027777

How I treat patients with AML using azacitidine and venetoclax

Andrew H. Wei, Sun Loo, Naval Daver Mar 20, 2025 pp. 1237-1250 10.1182/blood.2024024009

Abstract Venetoclax (VEN) received full approval in October 2020 for use in older patients who are unfit with acute myeloid leukemia (AML) combined with either hypomethylating agents...

Introduction to a How I Treat series on acute myeloid leukemia

Selina M. Luger Mar 20, 2025 pp. 1227-1228 10.1182/blood.2024027264

Treatment for patients with acute myeloid leukemia (AML) is being transformed for some patients, as targeted agents increase complete remission rates and survival when appropriately applied. Although...

How I treat acute myeloid leukemia with differentiation therapy

Ghayas C. Issa, Eytan M. Stein, Courtney D. DiNardo Mar 20, 2025 pp. 1251-1259 10.1182/blood.2024024008

Abstract An increasing number of acute myeloid leukemia (AML) therapeutics have been developed, not as cytotoxic therapies but rather as targeted agents able to restore the aberrant...

<i>Complement</i>ing CD20 antibodies’ effector functions

Jeanette Leusen, Thomas Valerius Mar 20, 2025 pp. 1232-1234 10.1182/blood.2024027767

How I treat secondary acute myeloid leukemia

Steven D. Green, Eunice S. Wang Mar 20, 2025 pp. 1260-1272 10.1182/blood.2024024011

Abstract Secondary acute myeloid leukemia (sAML) has traditionally been used to designate any AML disease arising from an antecedent hematologic disorder or after prior cytotoxic or...

LSS-Skin Sclerosis: key addition to cGVHD assessments

Najla El Jurdi, Steven Z. Pavletic Mar 20, 2025 pp. 1234-1235 10.1182/blood.2025028352

Potentiating CD20 monoclonal antibody therapy by targeting complement C3 fragments covalently deposited on lymphoma cells

Sivasubramanian Baskar, Haiyong Peng, Erika M. Gaglione et al. Mar 20, 2025 pp. 1309-1320 10.1182/blood.2024024846

Abstract Monoclonal antibodies (mAbs) improve survival of patients with mature B-cell malignancies. Fcγ receptor–dependent effector mechanisms kill tumor cells but can promote antige...

Development of the Lee Symptom Scale–Skin Sclerosis for chronic GVHD–associated sclerosis

Emily Baumrin, Joseph Pidala, Sandra A. Mitchell et al. Mar 20, 2025 pp. 1321-1332 10.1182/blood.2024027334

Abstract Sclerosis is a highly morbid manifestation of chronic graft-versus-host disease (cGVHD), associated with distressing symptoms and significant long-term disability. A patient...

Megakaryocyte mitotic figures in de novo AML, myelodysplasia related, with STAG2, SRSF2, and ASXL1 comutations

Zhaodong XU, Kristine Roland Mar 20, 2025 pp. 1333-1333 10.1182/blood.2024027590

How I use maintenance therapy in acute myeloid leukemia

Gail J. Roboz, Jonathan Canaani Mar 20, 2025 pp. 1273-1281 10.1182/blood.2024024010

Abstract Outcomes for acute myeloid leukemia (AML) have improved significantly in the past decade with the approval of novel therapeutics targeting diverse vulnerabilities of leukemi...

Triplets: the bright and the dark side of the moon

Anna Maria Frustaci, Alessandra Tedeschi Mar 20, 2025 pp. 1229-1231 10.1182/blood.2024028110

Zanubrutinib, venetoclax, and obinutuzumab in R/R CLL

Mar 20, 2025 pp. 1334-1334 10.1182/blood.2025028638

Population dynamics modeling reveals that myeloid bias involves both HSC differentiation and progenitor proliferation biases

Apeksha Singh, Jennifer J. Chia, Dinesh S. Rao et al. Mar 20, 2025 pp. 1293-1308 10.1182/blood.2024025598

Abstract Aging and chronic inflammation are associated with overabundant myeloid-primed multipotent progenitors (MPPs) among hematopoietic stem and progenitor cells (HSPCs). Although...

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Journal Info

Publisher Elsevier BV
ISSN 0006-4971
E-ISSN 1528-0020
Subject Health Sciences
Language English
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