Blood

Vol. 147, Issue 26 Issue 26 2026
16
Articles

Articles in this Issue

Diagnosis-to-treatment interval is associated with outcomes in follicular lymphoma treated with immunochemotherapy

Prokop Vodička, Tarec Christoffer El-Galaly, Vit Prochazka et al. Jun 25, 2026 pp. 3248-3252 10.1182/blood.2026033090

Abstract A short diagnosis-to-treatment interval independently predicts inferior outcomes in follicular lymphoma. Using international discovery and validation cohorts, we demonstra...

Beijing, Baltimore, or both: a randomized draw

David J. Curtis Jun 25, 2026 pp. 3125-3126 10.1182/blood.2026033922

CD49d: beyond a prognostic marker in CLL

Phuong-Hien Nguyen Jun 25, 2026 pp. 3126-3127 10.1182/blood.2026033672

Albumin-derived pseudo-Dutcher bodies in nonsecretory myeloma

Houlong Luo, Saihui Peng Jun 25, 2026 pp. 3253-3253 10.1182/blood.2026033553

Iron in HSCs: too much of a good thing

Massiel Chavez Stolla Jun 25, 2026 pp. 3131-3132 10.1182/blood.2026033923

A randomized trial of GVHD prophylaxis in haploidentical PBSC transplantation: ATG, PTCy, and low-dose combination therapy

Jun Yang, Yannan Jia, Xiaoxia Hu et al. Jun 25, 2026 pp. 3168-3178 10.1182/blood.2025032569

Abstract The optimal graft-versus-host disease (GVHD) prophylaxis strategy in haploidentical peripheral blood stem cell transplantation remains controversial. In this open-label, p...

Sutimlimab vs B-cell–targeted therapy in cold agglutinin disease: which is the optimal approach?

Bruno Fattizzo, Yoshitaka Miyakawa, Catherine Broome Jun 25, 2026 pp. 3135-3143 10.1182/blood.2026033150

Abstract Cold agglutinin disease (CAD) is a rare autoimmune hemolytic anemia caused by monoclonal immunoglobulin M autoantibodies that bind to red blood cells and trigger hemolysis...

Genotype-immunophenotype relationships in <i>NPM1</i> -mutated AML clonal evolution uncovered by single-cell multiomic analysis

Morgan Drucker, Darren Lee, Michael Bowman et al. Jun 25, 2026 pp. 3209-3216 10.1182/blood.2025030772

Abstract Acute myeloid leukemia (AML) is a multiclonal disease, existing as a milieu of clones with unique but related genotypes as initiating clones acquire subsequent mutations....

IRF2 is an essential transcription factor with pathogenic and prognostic impact in multiple myeloma

Nahia Gómez-Echarte, Arantxa Carrasco-Leon, Alba Maiques-Diaz et al. Jun 25, 2026 pp. 3195-3208 10.1182/blood.2025029422

Abstract Multiple myeloma (MM), the second most prevalent hematologic malignancy, remains incurable, highlighting the need to identify molecular drivers of disease progression and...

From bench to broken brains: freeze-dried platelets

Moritz Stolla Jun 25, 2026 pp. 3132-3133 10.1182/blood.2026033837

Single-cell tracking of the fittest in AML

Rachel Thijssen Jun 25, 2026 pp. 3129-3131 10.1182/blood.2026034221

In search of dependencies in myeloma, consider IRF2 too

Elisa Taiana, Niccolò Bolli Jun 25, 2026 pp. 3128-3129 10.1182/blood.2026033775

CD49d governs immune synapse formation through actin rearrangements and synchronizes BCR signaling in CLL

Laura Polcik, Abhishek Pethe, Driti Ashok et al. Jun 25, 2026 pp. 3179-3194 10.1182/blood.2024027753

Abstract B-cell receptor (BCR) signaling is a key determinant of chronic lymphocytic leukemia (CLL) pathophysiology. CD49d, the α4 subunit of the very late antigen 4 integrin, can...

A dried platelet–derived biologic for blood-brain barrier repair and hemorrhage control after TBI in mice

Alpa Trivedi, Byron Miyazawa, Haoqian Zhang et al. Jun 25, 2026 pp. 3231-3247 10.1182/blood.2025031826

Abstract Traumatic brain injury (TBI) is the leading cause of death in children and adults aged 1 to 44 years. Despite its high prevalence and devastating consequences, there are c...

Anti–HPA-1a fetal-neonatal alloimmune thrombocytopenia: reframing diagnostics, pathophysiology, and management

James B. Bussel, Jory M. Hirshman, Rick Kapur Jun 25, 2026 pp. 3144-3167 10.1182/blood.2025032550

Abstract Maternal alloantibodies directed to human platelet antigen 1a (HPA-1a) on fetal platelets can induce fetal-neonatal alloimmune thrombocytopenia (FNAIT), which causes intra...

Iron overload damages mitochondria and induces metabolic rewiring of hematopoietic stem cells toward glycolysis

Silvia Sighinolfi, Laura Cassina, Maria Rosa Lidonnici et al. Jun 25, 2026 pp. 3217-3230 10.1182/blood.2025031552

Abstract Iron is an essential element for most cellular processes, and recent evidence highlighted its role in regulating the function of hematopoietic stem cells (HSCs). Abnormal...

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Journal Info

Publisher Elsevier BV
ISSN 0006-4971
E-ISSN 1528-0020
Subject Health Sciences
Language English
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