Blood

Vol. 145, Issue 13 Issue 13 2025
15
Articles

Articles in this Issue

Warthin-Finkeldey cells seen in a setting other than measles: chronic lymphocytic leukemia/small lymphocytic lymphoma

Tanner J. Bakhshi, Sergio Pina-Oviedo Mar 27, 2025 pp. 1437-1437 10.1182/blood.2024028017

Sickle cell trait does not cause “sickle cell crisis” leading to exertion-related death: a systematic review

Lachelle D. Weeks, Allecia M. Wilson, Rakhi P. Naik et al. Mar 27, 2025 pp. 1345-1352 10.1182/blood.2024026899

Abstract Globally, an estimated 300 million individuals have sickle cell trait (SCT), the carrier state for sickle cell disease (SCD). Although SCD is associated with increased morbi...

Targeting an AML-sustaining metabolic node

Sarada Achyutuni, Jian Xu Mar 27, 2025 pp. 1341-1343 10.1182/blood.2024027768

Atypical chronic myeloid leukemia: from cytology to molecular characterization

Aida Calo-Pérez, Raquel Martínez-Fernández Mar 27, 2025 pp. 1438-1438 10.1182/blood.2024027505

Taming JAK cytokine release in haplo-transplant

Nikolas von Bubnoff Mar 27, 2025 pp. 1336-1338 10.1182/blood.2024027588

Reducing clinical trial eligibility barriers for patients with MDS: an icMDS position statement

Uma Borate, Kelly Pugh, Allyson Waller et al. Mar 27, 2025 pp. 1369-1381 10.1182/blood.2023023717

Abstract Excessively restrictive inclusion and exclusion criteria in clinical trials are one of many barriers to clinical trial enrollment for patients with myelodysplastic syndromes...

Two-layered immune escape in AML is overcome by Fcγ receptor activation and inhibition of PGE2 signaling in NK cells

Charlotte Rothfuß, Tobias Baumann, Sainitin Donakonda et al. Mar 27, 2025 pp. 1395-1406 10.1182/blood.2024025706

Abstract Loss of anticancer natural killer (NK) cell function in patients with acute myeloid leukemia (AML) is associated with fatal disease progression and remains poorly understood...

Jumping barriers in clinical trials: release the brake

Lionel Adès Mar 27, 2025 pp. 1335-1336 10.1182/blood.2024027711

Itacitinib for prevention of graft-versus-host disease and cytokine release syndrome in haploidentical transplantation

Ramzi Abboud, Mark A. Schroeder, Michael P. Rettig et al. Mar 27, 2025 pp. 1382-1394 10.1182/blood.2024026497

Abstract Haploidentical hematopoietic cell transplantation (haplo-HCT) is an increasingly used treatment for hematologic malignancies. Although posttransplant cyclophosphamide (PtCy)...

Two is better than one: dual targeting of WDR5 in T-ALL

Alexander A. Wurm, Denis M. Schewe Mar 27, 2025 pp. 1339-1341 10.1182/blood.2024027633

Impact of new medications on the treatment of immune TTP

Marie Scully, Lara Howells, William A. Lester Mar 27, 2025 pp. 1353-1357 10.1182/blood.2024026390

Abstract The last decade has seen the introduction of 2 new licensed therapies for thrombotic thrombocytopenic purpura (TTP), caplacizumab and recombinant ADAMTS13 (rADAMTS13), for i...

How I diagnose and treat acute infection–associated purpura fulminans

Pavan K. Bendapudi, Julie-Aurore Losman Mar 27, 2025 pp. 1358-1368 10.1182/blood.2024025078

Abstract Purpura fulminans (PF) is a rare but devastating complication of sepsis characterized by a highly thrombotic subtype of disseminated intravascular coagulation (DIC). A medic...

PGE2 inhibition to prevent AML escape from NK cells

Federico Simonetta Mar 27, 2025 pp. 1338-1339 10.1182/blood.2024027931

Targeting WDR5/ATAD2 signaling by the CK2/IKAROS axis demonstrates therapeutic efficacy in T-ALL

Qi Han, Yan Gu, Huimin Xiang et al. Mar 27, 2025 pp. 1407-1421 10.1182/blood.2024024130

Abstract T-cell acute lymphoblastic leukemia (T-ALL) is an aggressive hematological malignancy with a poor prognosis and limited options for targeted therapies. Identifying new molec...

α-Ketoglutarate dehydrogenase is a therapeutic vulnerability in acute myeloid leukemia

Scott E. Millman, Almudena Chaves-Perez, Sudha Janaki-Raman et al. Mar 27, 2025 pp. 1422-1436 10.1182/blood.2024025245

Abstract Perturbations in intermediary metabolism contribute to the pathogenesis of acute myeloid leukemia (AML) and can produce therapeutically actionable dependencies. Here, we p...

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Journal Info

Publisher Elsevier BV
ISSN 0006-4971
E-ISSN 1528-0020
Subject Health Sciences
Language English
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