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Daily briefing: Reflections from a survivor of the Hiroshima bombing
Predictions of Steady-State Photo-CIDNP Enhancement by Machine Learning
Cyano group translocation to alkenyl C(sp2)–H site by radical cation catalysis
Investigation of parallel joint density thresholds for granite tunnel failure based on physical model experiments and multiscale monitoring techniques
Isolation of Diamond Spin Chains in a Layered Halide Perovskite Heterostructure
Spin-polarized self-trapped excitons in low-dimensional cesium copper halide
Abstract Spin polarized excitons induced by spin injection from magnetic ion to a single quantum dot, has been considered as a basic unit of quantum information transfer between spin and photon for spin-photonic applications. However, this state-of-the-art technology has only been found with limited coupling strength and weak excitonic emission. Here, we demonstrate a spin-polarized self-trapped exciton naturally formed in the zero-dimensional lattice of cesium copper iodide. Upon excitation, the conversion from Cu+ ion to spin-1/2 Cu2+ ion results in an in-situ self-trapped exciton, which facilitates a local Jahn-Teller distortion and guarantees the strong spin-exciton coupling and near-unity excitonic emission efficiency. Consequently, a giant Zeeman splitting of −53 meV and an effective excitonic g-factor of −93.5 are observed from magneto-photoluminescence. More importantly, this nano-scale coupling can also be driven by an external electric field, which generates electroluminescence with a circular polarization of 44.5% at 4.2 K and 8% at 300 K. The spin-optic properties of this copper compound will stimulate the fabrication of next-generation spin-photonic devices based on self-trapped excitons.
Natural history study of recessive dystrophic epidermolysis bullosa wounds and patient reported outcomes using mobile application home photography
Symmetric Boron-Bridged Carbon Quantum Frameworks for Light-Emitting Diodes with over 20% External Quantum Efficiency
A genetically tractable non-vertebrate system to study complete camera-type eye regeneration
Integrated porosity estimation of partially saturated sand–hematite mixtures using electrical resistivity and elastic wave velocity
Breaking Bonds with Short-Wave Infrared Light: BODIPY Photocages for Two-Photon Activation in the 900–1500 nm NIR-II Window
Sequencing a DNA analog composed of artificial bases
Linking woody plant species richness with selected ecosystem services and dendrometric features in Okalma natural forest reserve
Dipole Characteristics of Polymers with Main-Chain Polar Rings: Impact of Dipole Strength and Orientation on Reactivity and Material Properties
Aging affects reprogramming of pulmonary capillary endothelial cells after lung injury in male mice
Abstract Aging increases the risk of developing fibrotic diseases by hampering tissue regeneration after injury. Using longitudinal single-cell RNA-seq and spatial transcriptomics, here we compare the transcriptome of bleomycin (BLM) -induced fibrotic lungs of young and aged male mice, at 3 time points corresponding to the peak of fibrosis, regeneration, and resolution. We find that lung injury shifts the transcriptomic profiles of three pulmonary capillary endothelial cells (PCEC) subpopulations. The associated signatures are linked to pro-angiogenic signaling with strong Lrg1 expression and do not progress similarly throughout the resolution process between young and old animals. Moreover, part of this set of resolution-associated markers is also detected in PCEC from samples of patients with idiopathic pulmonary fibrosis. Finally, we find that aging also alters the transcriptome of PCEC, which displays typical pro-fibrotic and pro-inflammatory features. We propose that age-associated alterations in specific PCEC subpopulations may interfere with the process of lung progenitor differentiation, thus contributing to the persistent fibrotic process typical of human pathology.