Iron-catalyzed active lipid peroxides drive ultrafast collective cell death in blooming algae

Y Yinjie Zhu (Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.) X Xiaoxiong Wang (Institute for Ocean Engineering & Center of Double Helix, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.) Y Yifan Tong (Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.) C Chengzhen Jia (Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.) H Huansheng Cao (Division of Natural and Applied Sciences, Duke Kunshan University, Kunshan, Jiangsu, China.) H Hongying Hu (Environmental Simulation and Pollution Control State Key Joint Laboratory, State Environmental Protection Key Laboratory of Microorganism Application and Risk Control (SMARC), School of Environment, Tsinghua University, Beijing, China.) Y Yi Tao (Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.)

Abstract

Harmful algal blooms, the most severe ecological hazards worldwide, terminate abruptly within a few days. In this work, we identified that iron-catalyzed active lipid peroxides predominantly trigger individual cell ferroptosis and drive the population collapse of blooming cyanobacteria. We reveal the chronological sequence of labile iron burst, oxidative stress, lipid peroxidation, and cell death during a Microcystis bloom demise event. Dead cells exhibit a nonrandom spatial distribution within colonies. Intensifying lipid peroxidation catalyzed by cellular labile iron generates truncated phospholipids with shortened fatty acyl chains bearing alkyl groups. These active lipid peroxides destabilize plasma membranes and induce nanoscale membrane pore formation, resulting in individual cell ferroptosis and lysis. Oxidized lipids are also released from ferroptotic cells, propagating lipid peroxidation to neighboring cells, thereby spreading death throughout the population.

Article Details

Journal Science
Volume / Issue Vol. 392, Issue 6805
Published June 25, 2026
ISSN 0036-8075
Publisher American Association for the Advancement of Science

Journal Info

Science

American Association for the Advancement of Science

ISSN: 0036-8075 Social Sciences

Authors (7)

Y

Yinjie Zhu

Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.

X

Xiaoxiong Wang

Institute for Ocean Engineering & Center of Double Helix, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.

Y

Yifan Tong

Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.

C

Chengzhen Jia

Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.

H

Huansheng Cao

Division of Natural and Applied Sciences, Duke Kunshan University, Kunshan, Jiangsu, China.

H

Hongying Hu

Environmental Simulation and Pollution Control State Key Joint Laboratory, State Environmental Protection Key Laboratory of Microorganism Application and Risk Control (SMARC), School of Environment, Tsinghua University, Beijing, China.

Y

Yi Tao

Guangdong Provincial Engineering Research Center for Urban Water Recycling and Environmental Safety, Tsinghua Shenzhen International Graduate School, Tsinghua University, Shenzhen, Guangdong, China.