WT EGFR and the oncogenic mutant EGFR (L858R) employ distinct mechanisms for export from the endoplasmic reticulum, a process critical for their activation

Mo Wang; Pik Ki Lau; Yusong Guo

doi:10.1016/j.jbc.2025.110312

WT EGFR and the oncogenic mutant EGFR (L858R) employ distinct mechanisms for export from the endoplasmic reticulum, a process critical for their activation

M Mo Wang P Pik Ki Lau (Department of Cellular and Molecular Medicine, University of California, San Diego School of Medicine, La Jolla, CA, USA.) Y Yusong Guo

View Original

APA MLA Chicago BibTeX RIS

Article Details

Journal Journal of Biological Chemistry

Volume / Issue Vol. 301, Issue 7

Published July 01, 2025

Pages 110312

DOI 10.1016/j.jbc.2025.110312

ISSN 0021-9258

Publisher Elsevier BV

Journal Info

Journal of Biological Chemistry

Elsevier BV

ISSN: 0021-9258 Open Access Life Sciences

Authors (3)

M

Mo Wang

P

Pik Ki Lau

Department of Cellular and Molecular Medicine, University of California, San Diego School of Medicine, La Jolla, CA, USA.

Y

Yusong Guo

WT EGFR and the oncogenic mutant EGFR (L858R) employ distinct mechanisms for export from the endoplasmic reticulum, a process critical for their activation

Article Details

Journal Info

Authors (3)

Related Articles from this Journal

Asialoglycoprotein receptor subunit Asgr1a loss results in attenuated cholesterol absorption in zebrafish fed a western diet

Nrf2-mediated epigenetic priming of osmoprotective genes enhances cellular adaptation to hyperosmotic stress

Saturated cardiolipins are potent disruptors of inner mitochondrial membrane structure and function

Epigenetic silencing of miR-141 via core promoter methylation is associated with short-term bladder cancer progression

Correction: Abstract 5588 Multifunctional carbon dots nanocarriers: Dual-drug delivery via π-π stacking interaction for stimuli-responsive chemotherapy and high oxidative stress