TGFβ links EBV to multisystem inflammatory syndrome in children

C Carl Christoph Goetzke M Mona Massoud S Stefan Frischbutter G Gabriela Maria Guerra M Marta Ferreira-Gomes F Frederik Heinrich A Anne Sae Lim von Stuckrad S Sebastian Wisniewski J Jan Robin Licha M Marina Bondareva L Lisa Ehlers S Samira Khaldi-Plassart E Etienne Javouhey S Sylvie Pons S Sophie Trouillet-Assant Y Yasemin Ozsurekci Y Yu Zhang (Xiangya Hospital, Central South University Changsha China) M Maria Cecilia Poli V Valentina Discepolo A Andrea Lo Vecchio B Bengü Sahin M Murielle Verboom M Michael Hallensleben A Anja Isabelle Heuhsen C Camila Astudillo Y Yazmin Espinosa M Maria Cecilia Vial Cox K Kerry Dobbs O Ottavia M. Delmonte G Gina A. Montealegre Sanchez M Mary Magliocco K Karyl Barron J Jeffrey Danielson L Lev Petrov N Nadine Unterwalder B Birgit Sawitzki M Mareen Matz K Katrin Lehmann A Alexander Gratopp H Horst von Bernuth L Lisa-Marie Burkhardt N Niklas Wiese L Lena Peter M Michael Schmueck-Henneresse L Leila Amini M Marcus Maurer J Jobst Fridolin Roehmel B Benjamin E. Gewurz L Lael M. Yonker M Mario Witkowski A Andrey Kruglov M Marcus Alexander Mall H Helen C. Su S Seza Ozen A Andreas Radbruch A Alexandre Belot P Pawel Durek T Tilmann Kallinich M Mir-Farzin Mashreghi

Abstract

Abstract In a subset of children and adolescents, SARS-CoV-2 infection induces a severe acute hyperinflammatory shock1 termed multisystem inflammatory syndrome in children (MIS-C) at four to eight weeks after infection. MIS-C is characterized by a specific T cell expansion2 and systemic hyperinflammation3. The pathogenesis of MIS-C remains largely unknown. Here we show that acute MIS-C is characterized by impaired reactivation of virus-reactive memory T cells, which depends on increased serum levels of the cytokine TGFβ resembling those that occur during severe COVID-19 (refs. 4,5). This functional impairment in T cell reactivity is accompanied by the presence of TGFβ-response signatures in T cells, B cells and monocytes along with reduced antigen-presentation capabilities of monocytes, and can be reversed by blocking TGFβ. Furthermore, T cell receptor repertoires of patients with MIS-C exhibit expansion of T cells expressing TCRVβ21.3, resembling Epstein–Barr virus (EBV)-reactive T cell clones capable of eliminating EBV-infected B cells. Additionally, serum TGFβ in patients with MIS-C can trigger EBV reactivation, which is reversible with TGFβ blockade. Clinically, the TGFβ-induced defect in T cell reactivity correlates with a higher EBV seroprevalence in patients with MIS-C compared with age-matched controls, along with the occurrence of EBV reactivation. Our findings establish a connection between SARS-CoV-2 infection and COVID-19 sequelae in children, in which impaired T cell cytotoxicity triggered by TGFβ overproduction leads to EBV reactivation and subsequent hyperinflammation.

Article Details

Journal Nature
Volume / Issue Vol. 640, Issue 8059
Published April 17, 2025
Pages 762-771
ISSN 0028-0836
Publisher Nature Portfolio

Journal Info

Nature

Nature Portfolio

ISSN: 0028-0836 Health Sciences

Authors (59)

C

Carl Christoph Goetzke

M

Mona Massoud

S

Stefan Frischbutter

G

Gabriela Maria Guerra

M

Marta Ferreira-Gomes

F

Frederik Heinrich

A

Anne Sae Lim von Stuckrad

S

Sebastian Wisniewski

J

Jan Robin Licha

M

Marina Bondareva

L

Lisa Ehlers

S

Samira Khaldi-Plassart

E

Etienne Javouhey

S

Sylvie Pons

S

Sophie Trouillet-Assant

Y

Yasemin Ozsurekci

Y

Yu Zhang

Xiangya Hospital, Central South University Changsha China

M

Maria Cecilia Poli

V

Valentina Discepolo

A

Andrea Lo Vecchio

B

Bengü Sahin

M

Murielle Verboom

M

Michael Hallensleben

A

Anja Isabelle Heuhsen

C

Camila Astudillo

Y

Yazmin Espinosa

M

Maria Cecilia Vial Cox

K

Kerry Dobbs

O

Ottavia M. Delmonte

G

Gina A. Montealegre Sanchez

M

Mary Magliocco

K

Karyl Barron

J

Jeffrey Danielson

L

Lev Petrov

N

Nadine Unterwalder

B

Birgit Sawitzki

M

Mareen Matz

K

Katrin Lehmann

A

Alexander Gratopp

H

Horst von Bernuth

L

Lisa-Marie Burkhardt

N

Niklas Wiese

L

Lena Peter

M

Michael Schmueck-Henneresse

L

Leila Amini

M

Marcus Maurer

J

Jobst Fridolin Roehmel

B

Benjamin E. Gewurz

L

Lael M. Yonker

M

Mario Witkowski

A

Andrey Kruglov

M

Marcus Alexander Mall

H

Helen C. Su

S

Seza Ozen

A

Andreas Radbruch

A

Alexandre Belot

P

Pawel Durek

T

Tilmann Kallinich

M

Mir-Farzin Mashreghi