Steroid therapy is linked to lower incidence of acute kidney injury in patients with severe alcohol-associated hepatitis

L Laura Buttler H Heiner Wedemeyer B Benjamin Maasoumy J Jan Stange D David Reich L Lance Stein N Nikolaos Pyrsopoulos V Valentin Cuervas-Mons R Raza Malik N Nikunj Shah S Simona Rossi J Juan Caballeria R Ram Subramanian A Andrés Duarte-Rojo J Julie Thompson P Peter R. Galle H Hartmut H.-J. Schmidt M Markus Busch (Technische Universität Darmstadt, Peter-Gruenberg-Strasse 8, Darmstadt 64289, Germany) K Kristina Chacko T Talal Adhami C Constance Mobley D David J. Kramer S Stephen Caldwell A Ali Al-Khafaji K Kalyan R. Bhamidimarri M Manuel Romero-Gomez T Tarek Hassanein W Waldo Concepcion M Martin Prieto Castillo R Rafael Bañares S Syed Naqvi M Matthias Dollinger K Karen Krok R Rudolf Stauber C Christian Zauner G Georg Lamprecht P Paul Thuluvath T Trinidad Serrano Aullo

Abstract

Abstract Acute kidney injury (AKI) is a common complication in patients with severe alcohol-associated hepatitis (sAH), yet its risk factors and outcomes remain incompletely defined. Corticosteroids are the only established therapy for sAH, but their impact on renal outcomes is unknown. A post-hoc analysis of the prospective multicenter VTL-308 trial including 151 patients with sAH was conducted to evaluate the incidence and predictors of AKI, the association of corticosteroid therapy with AKI risk, and factors associated with AKI reversal during 90 days of follow-up. AKI was independently associated with significantly higher 90-day mortality (subdistribution hazard ratio (sHR) = 8.74; p < 0.001). In multivariate competing risk analysis, higher bilirubin levels (sHR = 1.06; p = 0.003) were linked to increased AKI risk, while corticosteroids were associated with an ameliorated AKI risk (sHR = 0.47; p = 0.01). Time-censored analyses confirmed the protective association of corticosteroids (HR = 0.25; p = 0.001). Once AKI occurred, corticosteroids did not promote renal recovery (HR = 1.15; p = 0.74). Elevated bilirubin levels were linked to a lower probability of AKI reversal. In conclusion, AKI is a devastating complication in sAH. Corticosteroid therapy is independently associated with a significantly lower risk of AKI development but does not influence recovery once AKI has occurred. Future prospective trials are required to provide further validation of these findings.

Article Details

Volume / Issue Vol. 15, Issue 1
Published December 08, 2025
ISSN 2045-2322
Publisher Nature Portfolio

Journal Info

Scientific Reports

Nature Portfolio

ISSN: 2045-2322 Open Access Life Sciences

Authors (38)

L

Laura Buttler

H

Heiner Wedemeyer

B

Benjamin Maasoumy

J

Jan Stange

D

David Reich

L

Lance Stein

N

Nikolaos Pyrsopoulos

V

Valentin Cuervas-Mons

R

Raza Malik

N

Nikunj Shah

S

Simona Rossi

J

Juan Caballeria

R

Ram Subramanian

A

Andrés Duarte-Rojo

J

Julie Thompson

P

Peter R. Galle

H

Hartmut H.-J. Schmidt

M

Markus Busch

Technische Universität Darmstadt, Peter-Gruenberg-Strasse 8, Darmstadt 64289, Germany

K

Kristina Chacko

T

Talal Adhami

C

Constance Mobley

D

David J. Kramer

S

Stephen Caldwell

A

Ali Al-Khafaji

K

Kalyan R. Bhamidimarri

M

Manuel Romero-Gomez

T

Tarek Hassanein

W

Waldo Concepcion

M

Martin Prieto Castillo

R

Rafael Bañares

S

Syed Naqvi

M

Matthias Dollinger

K

Karen Krok

R

Rudolf Stauber

C

Christian Zauner

G

Georg Lamprecht

P

Paul Thuluvath

T

Trinidad Serrano Aullo