Pathogenic role of serpin B3-positive neutrophils in reinforcing thrombus stiffening during ischemic stroke

J Jiankun Zang (1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China) A Aijun Lu B Bing Yang N Na Tan (1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China) Q Qihuan Liu (1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China) L Liping Wei Y Ying Liang S Sijie Zhou Z Zefeng Tan (4Department of Neurology, The First People’s Hospital of Foshan, Foshan, China) X Xiufeng Xin (1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China) S Shengwen He (1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China) P PanWen Wu Y Yufeng Li Z Zhifeng Xu (6Department of Neurology, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China) X Xuanlin Su (1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China) H Hongcheng Mai (6Department of Neurology, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China) A Anding Xu (1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China) D Dan Lu (Institute for Carbon Neutralization Technology, College of Chemistry and Materials Engineering)

Abstract

Abstract The contribution of immune cells to thrombus architecture and mechanical properties in acute ischemic stroke (AIS) remains poorly understood. Using 3-dimensional imaging and multiplex staining, we mapped immune cells in human stroke thrombi and identified neutrophils as the dominant population. Analysis of 19 thrombi confirmed their positive correlation with collagen, increased stiffness, and poorer clinical outcomes. To preserve spatial context, we developed a laser capture–based proteomic workflow and analyzed thrombus neutrophils from 34 patients with AIS stratified by 90-day outcomes, followed by validation in an independent cohort of 22 patients. Proteomic analysis revealed serpin B3 as a neutrophil-enriched protein strongly correlated with poor prognosis. In murine models of ferric chloride–induced carotid artery thrombosis and middle cerebral artery occlusion, experiments using wild-type, neutrophil-depleted, and Serpinb3a knockout mice demonstrated that neutrophil-derived serpin B3 promotes early thrombus formation, enhances collagen deposition, and contributes to progressive thrombus stiffening. Mechanistically, serpin B3 secreted by neutrophils amplifies thrombus stiffness through upregulation of transforming growth factor β1, neutrophil extracellular traps, and COL1A1. Targeted Serpinb3a knockdown delayed vascular occlusion, improved thrombolysis efficiency, and resulted in better neurological recovery. Collectively, these findings identify a neutrophil-driven mechanism underlying thrombus stiffening and establish SERPINB3 as both a prognostic biomarker and a promising therapeutic target in AIS. This project has been registered with the Chinese Clinical Trial Registration Platform (https://www.chictr.org.cn/index.html) and has successfully passed the review process (registration number: ChiCTR2300077911).

Article Details

Journal Blood
Volume / Issue Vol. 147, Issue 25
Published June 18, 2026
Pages 3086-3101
ISSN 0006-4971
Publisher Elsevier BV

Journal Info

Blood

Elsevier BV

ISSN: 0006-4971 Health Sciences

Authors (18)

J

Jiankun Zang

1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China

A

Aijun Lu

B

Bing Yang

N

Na Tan

1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China

Q

Qihuan Liu

1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China

L

Liping Wei

Y

Ying Liang

S

Sijie Zhou

Z

Zefeng Tan

4Department of Neurology, The First People’s Hospital of Foshan, Foshan, China

X

Xiufeng Xin

1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China

S

Shengwen He

1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China

P

PanWen Wu

Y

Yufeng Li

Z

Zhifeng Xu

6Department of Neurology, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China

X

Xuanlin Su

1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China

H

Hongcheng Mai

6Department of Neurology, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, China

A

Anding Xu

1Department of Neurology and Stroke Center, The First Affiliated Hospital of Jinan University and State Key Laboratory of Bioactive Molecules and Druggability Assessment, Guangdong Basic Research Center of Excellence for Natural Bioactive Molecules and Discovery of Innovative Drugs, Jinan University, Guangzhou, China

D

Dan Lu

Institute for Carbon Neutralization Technology, College of Chemistry and Materials Engineering