Integrative spatial omics reveals distinct tumor-promoting multicellular niches and immunosuppressive mechanisms in Black American and White American patients with TNBC

Q Qian Zhu A Akhila Balasubramanian J Jaya Ruth Asirvatham M Megha Chatterjee B Badrajee Piyarathna J Jaspreet Kaur N Nada Mohamed L Ling Wu (State Key Lab of Photocatalysis on Energy and Environment, College of Chemistry) S Stacy Wang N Niloufar Pourfarrokh P Paula Danika Binsol M Mahak Bhargava U Uttam Rasaily Y Yitian Xu J Junjun Zheng D Deborah Jebakumar A Arundhati Rao C Carolina Gutierrez (Lester and Sue Smith Breast Center, Baylor College of Medicine) A Angela R. Omilian C Carl Morrison G Gokul M. Das C Christine Ambrosone E Erin H. Seeley S Shu-hsia Chen Y Yi Li E Eric Chang X Xiaoxian Li E Elizabeth Baker R Ritu Aneja X Xiang H.-F. Zhang (Lester and Sue Smith Breast Center, Baylor College of Medicine) A Arun Sreekumar (Department of Molecular and Cellular Biology, Baylor College of Medicine)

Abstract

Abstract Racial disparities in the clinical outcomes of triple-negative breast cancer (TNBC) have been well-documented, but the underlying biological mechanisms remain poorly understood. To investigate these disparities, we employed a multi-omic approach integrating imaging mass cytometry and spatial transcriptomics to characterize the tumor microenvironment (TME) in self-identified Black American (BA) and White American (WA) TNBC patients. Our analysis revealed that the TME in BA patients is marked by a network of endothelial cells, macrophages, and mesenchymal-like cells, which correlates with reduced patient survival. In contrast, the WA TNBC microenvironment is enriched in T-cells and neutrophils, indicative of T-cell exhaustion and suppressed immune responses. Ligand-receptor and pathway analyses further demonstrated that BA TNBC tumors exhibit a relatively “immune-cold” profile, while WA TNBC tumors display features of an “inflamed” TME, suggesting the evolution of a unique immunosuppressive mechanism. These findings provide insight into racially distinct tumor-promoting and immunosuppressive microenvironments, which may contribute to the observed differences in clinical outcomes among BA and WA TNBC patients.

Article Details

Volume / Issue Vol. 16, Issue 1
Published July 17, 2025
ISSN 2041-1723
Publisher Nature Portfolio

Journal Info

Nature Communications

Nature Portfolio

ISSN: 2041-1723 Open Access Life Sciences

Authors (31)

Q

Qian Zhu

A

Akhila Balasubramanian

J

Jaya Ruth Asirvatham

M

Megha Chatterjee

B

Badrajee Piyarathna

J

Jaspreet Kaur

N

Nada Mohamed

L

Ling Wu

State Key Lab of Photocatalysis on Energy and Environment, College of Chemistry

S

Stacy Wang

N

Niloufar Pourfarrokh

P

Paula Danika Binsol

M

Mahak Bhargava

U

Uttam Rasaily

Y

Yitian Xu

J

Junjun Zheng

D

Deborah Jebakumar

A

Arundhati Rao

C

Carolina Gutierrez

Lester and Sue Smith Breast Center, Baylor College of Medicine

A

Angela R. Omilian

C

Carl Morrison

G

Gokul M. Das

C

Christine Ambrosone

E

Erin H. Seeley

S

Shu-hsia Chen

Y

Yi Li

E

Eric Chang

X

Xiaoxian Li

E

Elizabeth Baker

R

Ritu Aneja

X

Xiang H.-F. Zhang

Lester and Sue Smith Breast Center, Baylor College of Medicine

A

Arun Sreekumar

Department of Molecular and Cellular Biology, Baylor College of Medicine