Identification of somatostatin <sup>+</sup> inhibitory engrams for extinction in the basolateral amygdala

X Xu Zhang Z Zhaohui Lan (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) Y Yu Huang Y Yuting Li (Division of Chemical and Biological Sciences) Z Zhenbo Wang (Key Laboratory of Regional Sustainable Development Modeling, Institute of Geographic Sciences and Natural Resources Research, Chinese Academy of Sciences) J Jingyu Sun (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) Y Ying Zhou H Hechen Bao (Department of Pharmacology, University of North Carolina) X Xuelian Fan (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) J Jiarun Yang (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) X Xiujuan Yang (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) C Caiqin Li (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) X Xiangyu Yang Y Yifang Kuang (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) M Miou Zhou (College of Dental Medicine, Western University of Health Sciences) G Guang He E Eiki Takahashi (Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University) P Paul W. Frankland W Weidong Li

Abstract

The basolateral amygdala (BLA) is a key structure for processing threat and emotional information, and plays a key role in controlling the fear memory. Previous research has suggested that the extinction procedure generates a new memory that coexists with the original fear memory, and that interneurons play an important role in this process. However, the mechanisms that control the competing extinction and fear memories in BLA are not yet fully understood. To investigate these mechanisms, we developed a chemogenetic strategy that offers improved sensitivity and specificity for tagging neurons during defined behavioral epochs. This was achieved by genetically targeting GABAergic interneurons for activity-dependent manipulation, allowing us to directly probe the functional role of inhibitory ensembles recruited during fear extinction. We found that silencing extinction-tagged BLA neurons or stimulating fear acquisition-tagged BLA neurons, led to a relapse of fear memory. Additionally, we observed that silencing BLA GABAergic neurons, or more specifically, silencing the extinction-tagged BLA GABAergic neurons, restored fear expression after extinction, while inhibiting acquisition-tagged BLA GABAergic neurons did not impair fear memory retrieval. Our results indicate that specific inhibitory GABAergic BLA engrams are established during fear extinction, which interfere with existing fear memory-related neural circuits and suppress conditioned fear memory. These findings provide insights into the neural mechanisms underlying fear extinction and suggest that BLA GABAergic neurons are potential targets for therapeutic interventions in cognitive disorders such as posttraumatic stress disorder (PTSD).

Article Details

Volume / Issue Vol. 123, Issue 12
Published March 24, 2026
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (19)

X

Xu Zhang

Z

Zhaohui Lan

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

Y

Yu Huang

Y

Yuting Li

Division of Chemical and Biological Sciences

Z

Zhenbo Wang

Key Laboratory of Regional Sustainable Development Modeling, Institute of Geographic Sciences and Natural Resources Research, Chinese Academy of Sciences

J

Jingyu Sun

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

Y

Ying Zhou

H

Hechen Bao

Department of Pharmacology, University of North Carolina

X

Xuelian Fan

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

J

Jiarun Yang

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

X

Xiujuan Yang

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

C

Caiqin Li

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

X

Xiangyu Yang

Y

Yifang Kuang

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

M

Miou Zhou

College of Dental Medicine, Western University of Health Sciences

G

Guang He

E

Eiki Takahashi

Bio-X Institutes, Key Laboratory for the Genetics of Development and Neuropsychiatric Disorders (Ministry of Education), Center for Brain Health and Brain Technology, Global Institute of Future Technology, Institute of Psychology and Behavioral Science, Shanghai Jiao Tong University

P

Paul W. Frankland

W

Weidong Li