Host ZAP activity correlates with the levels of CpG suppression in primate lentiviruses

R Rayhane Nchioua (Institute of Molecular Virology, Ulm University Medical Center) D Dorota Kmiec (Institute of Molecular Virology, Ulm University Medical Center) V Veronika Krchlikova (Research group "Mechanisms of innate Antiviral immunity", Institute for Medical Virology and Epidemiology of Viral Diseases, University Hospital Tübingen) S Sarah Mattes (Institute of Molecular Virology, Ulm University Medical Center) S Sabrina Noettger (Institute of Molecular Virology, Ulm University Medical Center) F Frederic Bibollet-Ruche R Ronnie M. Russell (Department of Medicine and Microbiology, Perelman School of Medicine, University of Pennsylvania) K Konstantin M. J. Sparrer (Institute of Molecular Virology) T Thomas Charpentier (Zoo La Palmyre) F Frédéric Tardy (Zoo Sanary-sur-Mer) S Steven E. Bosinger (Department of Pathology & Laboratory Medicine, Emory University, Division of Microbiology and Immunology, Emory National Primate Research Center) D Daniel Sauter (Research group "Mechanisms of innate Antiviral immunity", Institute for Medical Virology and Epidemiology of Viral Diseases, University Hospital Tübingen) B Beatrice H. Hahn F Frank Kirchhoff (Institute of Molecular Virology, Ulm University Medical Center)

Abstract

Zinc-finger antiviral protein (ZAP) is thought to drive the suppression of CpG dinucleotides in many viruses to mimic the composition of their host genomes. However, in vivo evidence is sparse. Here, we investigated the reasons for unusually high CpG levels in SIVmus and SIVmon from mustached and mona monkeys, descendants of one of the precursors of HIV-1. We show that SIVmus is not resistant to ZAP inhibition. Instead, these Cercopithecus monkey hosts differ from other primate species by a splice site mutation and express the poorly active extralarge XL rather than the highly active L isoform of ZAP. Similarly, higher CpG levels in endogenous prosimian lentiviruses were associated with low activity of the corresponding host lemur ZAPs. In addition, lemur genes also show lower CpG suppression than other primates. Thus, the antiviral activity of ZAP not only affects suppression of CpG dinucleotides in viral transcripts but possibly also host genomes.

Article Details

Volume / Issue Vol. 122, Issue 15
Published April 15, 2025
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (14)

R

Rayhane Nchioua

Institute of Molecular Virology, Ulm University Medical Center

D

Dorota Kmiec

Institute of Molecular Virology, Ulm University Medical Center

V

Veronika Krchlikova

Research group "Mechanisms of innate Antiviral immunity", Institute for Medical Virology and Epidemiology of Viral Diseases, University Hospital Tübingen

S

Sarah Mattes

Institute of Molecular Virology, Ulm University Medical Center

S

Sabrina Noettger

Institute of Molecular Virology, Ulm University Medical Center

F

Frederic Bibollet-Ruche

R

Ronnie M. Russell

Department of Medicine and Microbiology, Perelman School of Medicine, University of Pennsylvania

K

Konstantin M. J. Sparrer

Institute of Molecular Virology

T

Thomas Charpentier

Zoo La Palmyre

F

Frédéric Tardy

Zoo Sanary-sur-Mer

S

Steven E. Bosinger

Department of Pathology & Laboratory Medicine, Emory University, Division of Microbiology and Immunology, Emory National Primate Research Center

D

Daniel Sauter

Research group "Mechanisms of innate Antiviral immunity", Institute for Medical Virology and Epidemiology of Viral Diseases, University Hospital Tübingen

B

Beatrice H. Hahn

F

Frank Kirchhoff

Institute of Molecular Virology, Ulm University Medical Center