Glucocorticoid receptors in oligodendrocyte precursor cells regulate hippocampal network plasticity and stress-induced behavior in mice

L Lorenzo Mattioni (Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University) G Giulia Poggi (Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University) C Celine Gallagher (Institute of Physiology, University Medical Center of the Johannes Gutenberg-University) K Katrin Becker (Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University) L Linh Le (Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University) M Maja Papic (Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University) J Jasmin Engbers (Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University) M Maija-Kreetta Koskinen (SleepWell Research Program, Faculty of Medicine, University of Helsinki) A Ali Abdollahzadeh (A. I. Virtanen Institute for Molecular Sciences, University of Eastern Finland) D David P. Herzog (Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University) L Leonardo Nardi (Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University) A Andrea Conrad (Institute of Physiological Chemistry, University Medical Center of the Johannes Gutenberg University) S Sarah Winterberg (Department of Systemic Neuroscience, Institute of Anatomy and Cell Biology, Philipps University) C Christa Merte-Grebe (Department of Medical Cell Biology, Institute for Anatomy and Cell Biology, Medical Faculty, Philipps University Marburg) L Liana Melo-Thomas (Department of Systemic Neuroscience, Institute of Anatomy and Cell Biology, Philipps University) H Hyonseung Lee (Department of Systemic Neuroscience, Institute of Anatomy and Cell Biology, Philipps University) H Hans Schwarzbach (Department of Medical Cell Biology, Institute for Anatomy and Cell Biology, Medical Faculty, Philipps University Marburg) J Jennifer Klüpfel (Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University) R Ralf Kinscherf (Department of Medical Cell Biology, Institute for Anatomy and Cell Biology, Medical Faculty, Philipps University Marburg) J Jan Engelmann (Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University) B Beat Lutz (Leibniz Institute for Resilience Research) A Ari Waisman (Institute for Molecular Medicine and Research Center for Immunotherapy (FZI), University Medical Center of the Johannes Gutenberg University of Mainz, Mainz, Germany.) I Iiris Hovatta T Thomas Mittmann (Institute of Physiology, University Medical Center of the Johannes Gutenberg-University) M Michael J. Schmeisser (Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University) M Marianne B. Müller (Focus Program Translational Neurosciences, University Medical Center of the Johannes Gutenberg-University) G Giulia Treccani (Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University)

Abstract

Glucocorticoid receptors (GRs) are key mediators of how the stress hormone glucocorticoids (GCs) shape postnatal brain development and adaptive plasticity. Because GC signaling is critical during this period, postnatal GC concentrations are tightly regulated in the brain, whereas excessive levels of circulating GCs can disrupt developmental trajectories and increase the risk of psychiatric disorders later in life. GR function influences multiple neural cell types, but its cell-specific roles, particularly early in development, remain poorly understood. Oligodendrocyte precursor cells (OPCs), which generate myelinating oligodendrocytes and actively modulate neuronal networks, express GRs and can therefore respond to fluctuations in GC levels. Although excessive GC exposure during early life adversity has been linked to changes in OPC development, the physiological role of GR signaling specifically within OPCs remains unclear. To address this, we conditionally deleted GRs in postnatal OPCs in mice to investigate the role of physiological GC signaling in OPC proliferation and maturation, as well as in neuronal network activity and behavior. This deletion resulted in reduced oligodendrocyte and myelinated axon density in the hippocampus, sex-specific alterations in hippocampal activity and long-term potentiation following acute challenge, and impairments in memory formation in adulthood. Our findings reveal an OPC-specific role for GRs and suggest that physiological GR activity in the oligodendrocyte lineage contributes to normal hippocampal plasticity, learning, and memory.

Article Details

Volume / Issue Vol. 123, Issue 30
Published July 28, 2026
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (27)

L

Lorenzo Mattioni

Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University

G

Giulia Poggi

Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University

C

Celine Gallagher

Institute of Physiology, University Medical Center of the Johannes Gutenberg-University

K

Katrin Becker

Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University

L

Linh Le

Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University

M

Maja Papic

Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg-University

J

Jasmin Engbers

Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University

M

Maija-Kreetta Koskinen

SleepWell Research Program, Faculty of Medicine, University of Helsinki

A

Ali Abdollahzadeh

A. I. Virtanen Institute for Molecular Sciences, University of Eastern Finland

D

David P. Herzog

Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University

L

Leonardo Nardi

Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University

A

Andrea Conrad

Institute of Physiological Chemistry, University Medical Center of the Johannes Gutenberg University

S

Sarah Winterberg

Department of Systemic Neuroscience, Institute of Anatomy and Cell Biology, Philipps University

C

Christa Merte-Grebe

Department of Medical Cell Biology, Institute for Anatomy and Cell Biology, Medical Faculty, Philipps University Marburg

L

Liana Melo-Thomas

Department of Systemic Neuroscience, Institute of Anatomy and Cell Biology, Philipps University

H

Hyonseung Lee

Department of Systemic Neuroscience, Institute of Anatomy and Cell Biology, Philipps University

H

Hans Schwarzbach

Department of Medical Cell Biology, Institute for Anatomy and Cell Biology, Medical Faculty, Philipps University Marburg

J

Jennifer Klüpfel

Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University

R

Ralf Kinscherf

Department of Medical Cell Biology, Institute for Anatomy and Cell Biology, Medical Faculty, Philipps University Marburg

J

Jan Engelmann

Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University

B

Beat Lutz

Leibniz Institute for Resilience Research

A

Ari Waisman

Institute for Molecular Medicine and Research Center for Immunotherapy (FZI), University Medical Center of the Johannes Gutenberg University of Mainz, Mainz, Germany.

I

Iiris Hovatta

T

Thomas Mittmann

Institute of Physiology, University Medical Center of the Johannes Gutenberg-University

M

Michael J. Schmeisser

Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University

M

Marianne B. Müller

Focus Program Translational Neurosciences, University Medical Center of the Johannes Gutenberg-University

G

Giulia Treccani

Institute of Anatomy, University Medical Center of the Johannes Gutenberg-University