Germline variants in <i>UHRF1</i> are associated with multilocus imprinting disturbance in humans and mice

E Eguzkine Ochoa (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) I Ilona Zvetkova (Wellcome-Medical Research Council Institute of Metabolic Science and Medical Research Council Metabolic Diseases Unit, University of Cambridge) S Sunwoo Liv Lee (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) N Nozomi Takahashi (Department of Genetics, University of Cambridge) B Benoit Lan-Leung (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) E Emma Hobson (Leeds Clinical Genomics Service, Chapel Allerton Hospital, Leeds Teaching Hospitals National Health Service Trust) M Mahmoud Issa (Leeds Clinical Genomics Service, Chapel Allerton Hospital, Leeds Teaching Hospitals National Health Service Trust) B Bryndis Yngvadottir (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) F France Docquier (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) F Fay Rodger (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) D Dounia Foster-Hall (Department of Obstetrics and Gynaecology and National Institute for Health Research Cambridge Biomedical Research Centre) G Graeme Clark A Ana Toribio (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) E Ezequiel Martin (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) L Leonardo Bottolo (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre) A Anne C. Ferguson-Smith (Department of Genetics, University of Cambridge) W Wolfgang Fischle (Bioscience Program, Biological and Environmental Science and Engineering Division, King Abdullah University of Science and Technology) M Miguel Constancia (Wellcome-Medical Research Council Institute of Metabolic Science and Medical Research Council Metabolic Diseases Unit, University of Cambridge) E Eamonn R. Maher (Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre)

Abstract

The investigation of congenital imprinting disorders (CIDs) provides opportunities to elucidate the molecular mechanisms and role of genomic imprinting in development and human disease. Beckwith–Wiedemann spectrum (BWSp) is a prototypic CID resulting from genetic and epigenetic alterations of imprinted genes at chromosome 11p15.5. In up to a quarter of individuals with BWSp, the epigenetic alterations are not confined to 11p15.5 imprinting control regions but also involve other imprinted gene clusters (multilocus imprinting disturbance; MLID). In a consanguineous family with two children diagnosed with BWSp and MLID, the affected individuals were homozygous for a missense variant in UHRF1 , a gene previously implicated in the maintenance of DNA methylation. To investigate whether the UHRF1 c. 2001G&gt;C, p.(Lys667Asn) missense substitution predisposes to abnormal establishment/maintenance of genomic imprinting patterns, a genetically engineered mouse model with a Uhrf1 p.(Lys661Asn) variant was developed. Mice homozygous for the variant born to heterozygous mothers did not display an abnormal phenotype, but homozygotes born to healthy homozygous mothers displayed a range of phenotypes including prenatal lethality. Also, MLID was observed in affected mouse embryos. These findings are consistent with biallelic UHRF1 variants in affected individuals resulting in an autosomal recessively inherited cause of MLID in humans and expand the range of epigenetic disorders associated with UHRF1.

Article Details

Volume / Issue Vol. 122, Issue 34
Published August 26, 2025
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (19)

E

Eguzkine Ochoa

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

I

Ilona Zvetkova

Wellcome-Medical Research Council Institute of Metabolic Science and Medical Research Council Metabolic Diseases Unit, University of Cambridge

S

Sunwoo Liv Lee

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

N

Nozomi Takahashi

Department of Genetics, University of Cambridge

B

Benoit Lan-Leung

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

E

Emma Hobson

Leeds Clinical Genomics Service, Chapel Allerton Hospital, Leeds Teaching Hospitals National Health Service Trust

M

Mahmoud Issa

Leeds Clinical Genomics Service, Chapel Allerton Hospital, Leeds Teaching Hospitals National Health Service Trust

B

Bryndis Yngvadottir

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

F

France Docquier

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

F

Fay Rodger

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

D

Dounia Foster-Hall

Department of Obstetrics and Gynaecology and National Institute for Health Research Cambridge Biomedical Research Centre

G

Graeme Clark

A

Ana Toribio

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

E

Ezequiel Martin

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

L

Leonardo Bottolo

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre

A

Anne C. Ferguson-Smith

Department of Genetics, University of Cambridge

W

Wolfgang Fischle

Bioscience Program, Biological and Environmental Science and Engineering Division, King Abdullah University of Science and Technology

M

Miguel Constancia

Wellcome-Medical Research Council Institute of Metabolic Science and Medical Research Council Metabolic Diseases Unit, University of Cambridge

E

Eamonn R. Maher

Department of Genomic Medicine, University of Cambridge and National Institute for Health and Care Research Cambridge Biomedical Research Centre