Dysregulated NAMPT signaling underlines the immune-suppressive microenvironment in venous leg ulcers

J Jiating Wang (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) L Lei Guo (Quantitative Biomedical Research Center, Department of Health Science & Biostatistics, Peter O’Donnell Jr. School of Public Health, University of Texas Southwestern Medical Center, Dallas, TX, USA.) Y Yunxi Tao (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) Y Yunting Xiao (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) Y Yejing Huang (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) L Ling Pan (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) C Chen Lyu (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) L Liping Zhu (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) X Xiya Zhang (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) J Juan Gao (Institute of Soil Science, Chinese Academy of Sciences , ,) C Chunyan Cao (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) Z Zhuang Liu (Macao Institute of Materials Science and Engineering) N Ning Xu Landén (Dermatology and Venereology Division, Department of Medicine Solna, Center for Molecular Medicine, Karolinska Institutet) J Jing Wang (Hunan Cancer Hospital Changsha China) T Tongbin Chu (Department of Wound Regeneration, The Second Hospital of Dalian Medical University) X Xinfeng Wu (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) H Hongsheng Wang (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College) D Dongqing Li (Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College)

Abstract

Venous leg ulcers (VLUs) are the most common form of lower extremity ulceration, with a rising prevalence among the elderly. Despite their clinical significance, the molecular mechanisms underlying VLU remain poorly defined. In this study, we constructed a comprehensive single-cell transcriptional atlas of VLUs, unveiling a widespread immunosuppressive microenvironment within the lesions. Specifically, stromal cells exhibited attenuated inflammatory responses coupled with enhanced fibrotic activity. Keratinocytes and endothelial cells demonstrated increased proliferative and angiogenic activity, respectively, yet both cell types showed markedly reduced antigen-presenting capacity. Immune cell compartments displayed profound dysfunction characterized by marked reductions and impaired functionality of dendritic cells, polarization of macrophages toward an anti-inflammatory M2 phenotype, and enrichment of T cell exhaustion signatures. Notably, cell–cell communication analysis revealed diminished crosstalk between immune and structural cell populations. Mechanistically, we identified NAMPT-mediated signaling as a critical regulator of myeloid-stromal crosstalk. Targeted depletion of NAMPT in macrophages disrupted fibroblast-mediated inflammatory responses and impaired wound healing, whereas exogenous NAMPT administration reactivated immune responses and promoted tissue repair in chronic wounds. Collectively, these findings provide a comprehensive molecular framework for understanding immune–stromal dysfunction in VLUs and position NAMPT as a central immune regulator and promising therapeutic target for chronic wound treatment.

Article Details

Volume / Issue Vol. 122, Issue 52
Published December 30, 2025
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (18)

J

Jiating Wang

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

L

Lei Guo

Quantitative Biomedical Research Center, Department of Health Science & Biostatistics, Peter O’Donnell Jr. School of Public Health, University of Texas Southwestern Medical Center, Dallas, TX, USA.

Y

Yunxi Tao

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

Y

Yunting Xiao

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

Y

Yejing Huang

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

L

Ling Pan

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

C

Chen Lyu

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

L

Liping Zhu

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

X

Xiya Zhang

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

J

Juan Gao

Institute of Soil Science, Chinese Academy of Sciences , ,

C

Chunyan Cao

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

Z

Zhuang Liu

Macao Institute of Materials Science and Engineering

N

Ning Xu Landén

Dermatology and Venereology Division, Department of Medicine Solna, Center for Molecular Medicine, Karolinska Institutet

J

Jing Wang

Hunan Cancer Hospital Changsha China

T

Tongbin Chu

Department of Wound Regeneration, The Second Hospital of Dalian Medical University

X

Xinfeng Wu

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

H

Hongsheng Wang

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College

D

Dongqing Li

Jiangsu Provincial Key Laboratory of Dermatology, Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences & Peking Union Medical College