Chronic infection perturbs the affinity hierarchy of antiviral B cells

M Mirela Dimitrova (Department of Biomedicine, Division of Experimental Virology, University of Basel) T Tiago Abreu-Mota (Department of Biomedicine, Division of Experimental Virology, University of Basel) J Jonas Fixemer (Department of Biomedicine, Division of Experimental Virology, University of Basel) W Weldy V. Bonilla (Department of Biomedicine, Division of Experimental Virology, University of Basel) A Anna-Friederike Marx (Department of Biomedicine, Division of Experimental Virology, University of Basel) M Min Lu (College of Chemistry and Chemical Engineering, Frontiers Science Center for Rare Isotopes) K Karen Tintignac (Department of Biomedicine, Division of Experimental Virology, University of Basel) A Anna Lena Kastner (Department of Biomedicine, Division of Experimental Virology, University of Basel) Y Yusuf I. Ertuna (Department of Biomedicine, Division of Experimental Virology, University of Basel) M Marianna Florova (Department of Biomedicine, Division of Experimental Virology, University of Basel) M Matias Ciancaglini (Department of Biomedicine, Division of Experimental Virology, University of Basel) K Kerstin Narr (Department of Biomedicine, Division of Experimental Virology, University of Basel) K Karsten Stauffer (Department of Biomedicine, Division of Experimental Virology, University of Basel) J Julien Roux (Department of Biomedicine, Bioinformatics Core Facility, University Hospital Basel) P Philippe Demougin (Genomics Facility Basel, Pharmacenter, University of Basel) I Ingrid Wagner (Department of Pathology and Immunology, University of Geneva) D Doron Merkler (Department of Pathology and Immunology, Faculty of Medicine, University of Geneva) D Daniel D. Pinschewer (Department of Biomedicine, Division of Experimental Virology, University of Basel)

Abstract

A key function of the germinal center (GC) reaction consists in the preferential expansion and enrichment of high-affinity B cell clones. Whether and how persistent viral infection thwarts this purpose remains ill-defined. Here, we transferred monoclonal lymphocytic choriomeningitis virus–specific B cells into persistently infected hosts. High-affinity B cells expanded vigorously, forming GCs and abundant antibody-secreting cells. When failing to clear viremia or to drive viral mutational escape, the expanded B cell population contracted, ending in its quasi-complete disappearance from the spleen, a process we termed “attrition.” In stark contrast, low-affinity B cells expanded and persisted irrespective of high viral loads. B cell attrition was associated with phenotypic and transcriptional alterations including a prominent Blimp-1 transcriptional signature in high-affinity GC B cells. Blimp-1-deficient B cells were resistant to attrition, suggesting a B cell–intrinsic process. Moreover, exogenously supplied antibody feedback prevented attrition, suggesting that the latter may have resulted from excessive antigenic stimulation. Our findings suggest that in chronic viral infection, the incessant exposure to overwhelming amounts of antigen perturbs B cell affinity hierarchies by preferentially dysregulating high-affinity B cells.

Article Details

Volume / Issue Vol. 123, Issue 14
Published April 07, 2026
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (18)

M

Mirela Dimitrova

Department of Biomedicine, Division of Experimental Virology, University of Basel

T

Tiago Abreu-Mota

Department of Biomedicine, Division of Experimental Virology, University of Basel

J

Jonas Fixemer

Department of Biomedicine, Division of Experimental Virology, University of Basel

W

Weldy V. Bonilla

Department of Biomedicine, Division of Experimental Virology, University of Basel

A

Anna-Friederike Marx

Department of Biomedicine, Division of Experimental Virology, University of Basel

M

Min Lu

College of Chemistry and Chemical Engineering, Frontiers Science Center for Rare Isotopes

K

Karen Tintignac

Department of Biomedicine, Division of Experimental Virology, University of Basel

A

Anna Lena Kastner

Department of Biomedicine, Division of Experimental Virology, University of Basel

Y

Yusuf I. Ertuna

Department of Biomedicine, Division of Experimental Virology, University of Basel

M

Marianna Florova

Department of Biomedicine, Division of Experimental Virology, University of Basel

M

Matias Ciancaglini

Department of Biomedicine, Division of Experimental Virology, University of Basel

K

Kerstin Narr

Department of Biomedicine, Division of Experimental Virology, University of Basel

K

Karsten Stauffer

Department of Biomedicine, Division of Experimental Virology, University of Basel

J

Julien Roux

Department of Biomedicine, Bioinformatics Core Facility, University Hospital Basel

P

Philippe Demougin

Genomics Facility Basel, Pharmacenter, University of Basel

I

Ingrid Wagner

Department of Pathology and Immunology, University of Geneva

D

Doron Merkler

Department of Pathology and Immunology, Faculty of Medicine, University of Geneva

D

Daniel D. Pinschewer

Department of Biomedicine, Division of Experimental Virology, University of Basel