Characterization of KRASG12C inhibitor olomorasib single-agent and combination with activity in KRASG12C-mutant models

S Shengbin Peng Y Youyan Zhang X Xi Lin C Chong Si R Robert Daniel Van Horn J Jack A. Dempsey E Eva Goetz R Robert J. Evans A Andrew Farber M Matthew J. Vandekopple W Wenyu Ming H Hong Gao (Beijing National Laboratory for Molecular Sciences (BNLMS), Institute of Chemistry) C Chungping Yu W Wei Guo Xu N Nicholas E. Brown M Michele S. Dowless N Nicholas Pulliam D David A. Barda D Deqi Guo S Serge L. Boulet L Lysian Huber A Andrew Capen B Bonita Jones S Sarah Bogner M Mark A. Castanares J Jennifer Rachelle Stephens M Megan A. Johnson C Carmen L. Curtis J John M. Strelow J Junpeng Xiao J Josh Ballard W Wayne P. Bocchinfuso M Michael J. Chalmers J Jing Wang (Hunan Cancer Hospital Changsha China) J Jorg Hendle M Melbert D. Saflor D Danalyn Manglicmot Lagutan T Tarun Gheyi A Anita Sarkar M Margaret Kearins F Frances Tung J Joseph Ho L Logan Rodgers J Jordi Benach A Anton Joseph Frommelt L Lian Zhou B Bradley L. Ackermann D Denis McCann A Anke Klippel S Sean G. Buchanan J James R. Henry X Xueqian Gong

Abstract

Abstract The impact of first-generation covalent KRAS G12C inhibitors has been reduced due to the development of drug resistance, tolerability and challenges combining with immunotherapy. We designed olomorasib, a next-generation GDP-binding KRAS G12C inhibitor, for nanomolar potency as well as selectivity over wild-type inhibition. In both in vitro and in vivo models of KRAS G12C -mutant cancers, olomorasib reduces RAS activity and pERK levels, leading to substantial and significant tumor growth inhibition. Additionally, olomorasib combined with immune checkpoint inhibitors demonstrates greater anti-tumor activity compared to monotherapy. Furthermore, we demonstrate that olomorasib binds tightly to KRAS G12C even in the presence of clinically relevant second site mutations, a known mechanism of resistance and limitation to currently approved KRAS G12C inhibitors. These findings suggest that olomorasib could be effective for patients with KRAS G12C mutant cancers either as monotherapy or in combination with immunotherapy. Olomorasib monotherapy and combination treatments are currently being investigated clinically.

Article Details

Volume / Issue Vol. 17, Issue 1
Published May 04, 2026
ISSN 2041-1723
Publisher Nature Portfolio

Journal Info

Nature Communications

Nature Portfolio

ISSN: 2041-1723 Open Access Life Sciences

Authors (52)

S

Shengbin Peng

Y

Youyan Zhang

X

Xi Lin

C

Chong Si

R

Robert Daniel Van Horn

J

Jack A. Dempsey

E

Eva Goetz

R

Robert J. Evans

A

Andrew Farber

M

Matthew J. Vandekopple

W

Wenyu Ming

H

Hong Gao

Beijing National Laboratory for Molecular Sciences (BNLMS), Institute of Chemistry

C

Chungping Yu

W

Wei Guo Xu

N

Nicholas E. Brown

M

Michele S. Dowless

N

Nicholas Pulliam

D

David A. Barda

D

Deqi Guo

S

Serge L. Boulet

L

Lysian Huber

A

Andrew Capen

B

Bonita Jones

S

Sarah Bogner

M

Mark A. Castanares

J

Jennifer Rachelle Stephens

M

Megan A. Johnson

C

Carmen L. Curtis

J

John M. Strelow

J

Junpeng Xiao

J

Josh Ballard

W

Wayne P. Bocchinfuso

M

Michael J. Chalmers

J

Jing Wang

Hunan Cancer Hospital Changsha China

J

Jorg Hendle

M

Melbert D. Saflor

D

Danalyn Manglicmot Lagutan

T

Tarun Gheyi

A

Anita Sarkar

M

Margaret Kearins

F

Frances Tung

J

Joseph Ho

L

Logan Rodgers

J

Jordi Benach

A

Anton Joseph Frommelt

L

Lian Zhou

B

Bradley L. Ackermann

D

Denis McCann

A

Anke Klippel

S

Sean G. Buchanan

J

James R. Henry

X

Xueqian Gong