Beyond genotype: challenges in predicting disease risk for carriers of biallelic perforin variants

O Oliver Wegehaupt (1Institute for Immunodeficiency, Center for Chronic Immunodeficiency, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany) O Oleg Borisov (3Institute of Genetic Epidemiology, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany) E Elena Sieni (8Azienda Ospedaliera Universitaria Meyer, Firenze, Italy) F Florian Oyen (5Division of Pediatric Stem Cell Transplantation and Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany) J Jasmin Mann (1Institute for Immunodeficiency, Center for Chronic Immunodeficiency, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany) M Maria Luisa Coniglio (4Pediatric Hematology Oncology, Meyer Children’s Hospital IRCCS, Florence, Italy) A Aurora Chinnici (4Pediatric Hematology Oncology, Meyer Children’s Hospital IRCCS, Florence, Italy) F Francesco Pegoraro (2Department of experimental and clinical medicine, Florence, Italy) L Linda Beneforti (4Pediatric Hematology Oncology, Meyer Children’s Hospital IRCCS, Florence, Italy) K Kimberly Gilmour (Great Ormond Street Hospital for Children NHS Trust, London) D Despina Moshous (8Université Paris Cité, Pediatric Hematology-Immunology and Rheumatology Department, Necker-Enfants Malades Hospital, Paris, France, Paris, France) G Geneviève de Saint Basile (9Imagine Institute, Université de Paris Cité, INSERM U1163, Paris, France) W Wenying Zhang (State Key Laboratory of Rare Earth Resource Utilization) R Rebecca Marsh (11Department of Pediatrics, University of Cincinnati, College of Medicine, Cincinnati, OH) C Carmela De Fusco (14Department of Oncology, Hematology, and Cell Therapy, AORN Santobono Pausilipon, Naples, Italy) K Katharina Wustrau (14Department of pediatrics, University Medical Center Ulm, Ulm, Germany, Ulm, Germany) F Fabio Timeus (16Pediatrics Department, Chivasso Hospital, Turin, Italy) C Concetta Micalizzi (18Pediatric Hematology Oncology, IRCCS Istituto Giannina Gaslini, Genoa, Italy) E Eberhard Gunsilius (19Department of Hematology and Oncology, Medical University Innsbruck, Innsbruck, Austria) L Laine Hosking (20Department of Allergy and Immunology, The Royal Children's Hospital, Melbourne, VIC, Australia) S Sharon Choo (20Department of Allergy and Immunology, The Royal Children's Hospital, Melbourne, VIC, Australia) S Sujal Ghosh (21Department of Pediatric Oncology, Hematology, and Clinical Immunology, Center of Child and Adolescent Health, Heinrich Heine University, Duesseldorf, Germany) A Anna Köttgen K Kai Lehmberg (5Division of Pediatric Stem Cell Transplantation and Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany) S Stephan Ehl (1Institute for Immunodeficiency, Center for Chronic Immunodeficiency, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany)

Abstract

Abstract Genetic screening for severe congenital immunohematological diseases offers potential for early intervention, particularly through preemptive allogeneic hematopoietic stem cell transplantation (HSCT). However, the clinical value of such screening depends on precise prognostic predictions based on genotype-phenotype correlations and/or functional confirmation. We investigated familial hemophagocytic lymphohistiocytosis type 2 (FHL2), caused by PRF1 variants. Specifically, we evaluated the clinical significance of the frequent PRF1 A91V variant, if present in trans with a predicted loss-of-function (pLOF) PRF1 variant, defined as “disease mutation” listed in the Human Gene Mutation Database. We combined clinical and functional data from our hemophagocytic lymphohistiocytosis (HLH)–network registry with UK Biobank data to evaluate disease penetrance and clinical outcomes. Among 52 individuals with A91V/pLOF genotype in the registry, 39 (72%) showed FHL2-related manifestations with mean onset at 20 years. Four patients had recurrent disease, 15 received transplantation, and 14 died. Among 14 individuals with A91V/pLOF genotype identified by family screening (mean age, 29 years), however, only 1 was symptomatic. Moreover, among 21 A91V/pLOF carriers identified in 200 000 UK Biobank participants, 12 with genotypes identical to symptomatic registry patients, none had developed HLH by age 73 years. Premature stop pLOF alleles appeared more penetrant than missense variants, but functional data including perforin expression or cytotoxicity failed to predict disease manifestation. Our combined registry and population-based approach reveals significant variability in disease penetrance and severity among PRF1 A91V/pLOF carriers, with no clear association between genotype, functional data, and clinical outcomes. This complexity illustrates the challenges of genetic screening and highlights the need for careful clinical decision-making regarding preemptive HSCT in asymptomatic carriers.

Article Details

Journal Blood
Volume / Issue Vol. 145, Issue 25
Published June 19, 2025
Pages 2992-3006
ISSN 0006-4971
Publisher Elsevier BV

Journal Info

Blood

Elsevier BV

ISSN: 0006-4971 Health Sciences

Authors (25)

O

Oliver Wegehaupt

1Institute for Immunodeficiency, Center for Chronic Immunodeficiency, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany

O

Oleg Borisov

3Institute of Genetic Epidemiology, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany

E

Elena Sieni

8Azienda Ospedaliera Universitaria Meyer, Firenze, Italy

F

Florian Oyen

5Division of Pediatric Stem Cell Transplantation and Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany

J

Jasmin Mann

1Institute for Immunodeficiency, Center for Chronic Immunodeficiency, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany

M

Maria Luisa Coniglio

4Pediatric Hematology Oncology, Meyer Children’s Hospital IRCCS, Florence, Italy

A

Aurora Chinnici

4Pediatric Hematology Oncology, Meyer Children’s Hospital IRCCS, Florence, Italy

F

Francesco Pegoraro

2Department of experimental and clinical medicine, Florence, Italy

L

Linda Beneforti

4Pediatric Hematology Oncology, Meyer Children’s Hospital IRCCS, Florence, Italy

K

Kimberly Gilmour

Great Ormond Street Hospital for Children NHS Trust, London

D

Despina Moshous

8Université Paris Cité, Pediatric Hematology-Immunology and Rheumatology Department, Necker-Enfants Malades Hospital, Paris, France, Paris, France

G

Geneviève de Saint Basile

9Imagine Institute, Université de Paris Cité, INSERM U1163, Paris, France

W

Wenying Zhang

State Key Laboratory of Rare Earth Resource Utilization

R

Rebecca Marsh

11Department of Pediatrics, University of Cincinnati, College of Medicine, Cincinnati, OH

C

Carmela De Fusco

14Department of Oncology, Hematology, and Cell Therapy, AORN Santobono Pausilipon, Naples, Italy

K

Katharina Wustrau

14Department of pediatrics, University Medical Center Ulm, Ulm, Germany, Ulm, Germany

F

Fabio Timeus

16Pediatrics Department, Chivasso Hospital, Turin, Italy

C

Concetta Micalizzi

18Pediatric Hematology Oncology, IRCCS Istituto Giannina Gaslini, Genoa, Italy

E

Eberhard Gunsilius

19Department of Hematology and Oncology, Medical University Innsbruck, Innsbruck, Austria

L

Laine Hosking

20Department of Allergy and Immunology, The Royal Children's Hospital, Melbourne, VIC, Australia

S

Sharon Choo

20Department of Allergy and Immunology, The Royal Children's Hospital, Melbourne, VIC, Australia

S

Sujal Ghosh

21Department of Pediatric Oncology, Hematology, and Clinical Immunology, Center of Child and Adolescent Health, Heinrich Heine University, Duesseldorf, Germany

A

Anna Köttgen

K

Kai Lehmberg

5Division of Pediatric Stem Cell Transplantation and Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany

S

Stephan Ehl

1Institute for Immunodeficiency, Center for Chronic Immunodeficiency, Medical Center, Faculty of Medicine, University of Freiburg, Freiburg, Germany