A preclinical pig model of Angelman syndrome mirrors the early developmental trajectory of the human condition

L Luke S. Myers (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) S Sarah G. Christian (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) S Sean Simpson (Department of Molecular Biomedical Sciences, College of Veterinary Medicine, North Carolina State University) R Renan Sper (Department of Molecular Biomedical Sciences, College of Veterinary Medicine, North Carolina State University) C Clint Taylor (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) L Laura Montes (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) T Thomas B. C. Jepp (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) D Daniela Ramos (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) L Livia Schuller (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) K Kranti Konganti (Texas A&M University Institute for Genome Sciences and Society, Texas A&M University) W Wade Friedeck (Veterinary Medical Teaching Hospital, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) O Ozair Habib (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) M McKaela Hodge (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) A Alasdair J. Taylor (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) A Ashley Coffell (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) A Annalise Schlafer (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) M Morgan Matt (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) B Bradley Revell (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) C Carol Knight (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) C Cristina Cardona-Barreña (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University) W William J. Murphy E Edwin J. Weeber (Department of Molecular Pharmacology and Physiology, University of South Florida) D David J. Segal (MIND Institute, University of California Davis School of Medicine) A Anne Anderson (Department of Pediatrics, Baylor College of Medicine) K Kevin R. Nash (Department of Molecular Pharmacology and Physiology, University of South Florida) J Jill L. Silverman (MIND Institute, University of California Davis School of Medicine) J Jorge A. Piedrahita (Department of Molecular Biomedical Sciences, College of Veterinary Medicine, North Carolina State University) S Scott V. Dindot (Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University)

Abstract

Angelman syndrome is a neurodevelopmental disorder characterized by severe motor and cognitive deficits. It is caused by the loss of the maternally inherited allele of the imprinted ubiquitin-protein ligase E3A ( UBE3A ) gene. Rodent models of Angelman syndrome do not fully recapitulate all the symptoms associated with the condition and are limited as a preclinical model for therapeutic development. Here, we show that pigs ( Sus scrofa ) with a maternally inherited deletion of UBE3A ( UBE3A -/+ ) have altered postnatal behaviors, impaired vocalizations, reduced brain growth, motor incoordination, and ataxia. Neonatal UBE3A -/+ pigs exhibited several symptoms observed in infants with Angelman syndrome, including hypotonia, suckling deficits, and failure to thrive. Collectively, these findings are consistent with the pathophysiology and developmental trajectory observed in individuals with Angelman syndrome. We anticipate that this pig model will advance our understanding of the pathophysiology of Angelman syndrome and be used as a preclinical large animal model for therapeutic development.

Article Details

Volume / Issue Vol. 122, Issue 30
Published July 29, 2025
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (28)

L

Luke S. Myers

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

S

Sarah G. Christian

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

S

Sean Simpson

Department of Molecular Biomedical Sciences, College of Veterinary Medicine, North Carolina State University

R

Renan Sper

Department of Molecular Biomedical Sciences, College of Veterinary Medicine, North Carolina State University

C

Clint Taylor

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

L

Laura Montes

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

T

Thomas B. C. Jepp

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

D

Daniela Ramos

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

L

Livia Schuller

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

K

Kranti Konganti

Texas A&M University Institute for Genome Sciences and Society, Texas A&M University

W

Wade Friedeck

Veterinary Medical Teaching Hospital, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

O

Ozair Habib

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

M

McKaela Hodge

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

A

Alasdair J. Taylor

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

A

Ashley Coffell

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

A

Annalise Schlafer

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

M

Morgan Matt

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

B

Bradley Revell

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

C

Carol Knight

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

C

Cristina Cardona-Barreña

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University

W

William J. Murphy

E

Edwin J. Weeber

Department of Molecular Pharmacology and Physiology, University of South Florida

D

David J. Segal

MIND Institute, University of California Davis School of Medicine

A

Anne Anderson

Department of Pediatrics, Baylor College of Medicine

K

Kevin R. Nash

Department of Molecular Pharmacology and Physiology, University of South Florida

J

Jill L. Silverman

MIND Institute, University of California Davis School of Medicine

J

Jorge A. Piedrahita

Department of Molecular Biomedical Sciences, College of Veterinary Medicine, North Carolina State University

S

Scott V. Dindot

Department of Veterinary Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University