A µ-opioid receptor superagonist analgesic with minimal adverse effects

J Juan L. Gomez E Emilya N. Ventriglia Z Zachary J. Frangos A Agnieszka Sulima M Michael J. Robertson M Michael D. Sacco R Reece C. Budinich I Ilinca M. Giosan T Tongzhen Xie O Oscar Solis A Anna E. Tischer J Jennifer M. Bossert K Kiera E. Caldwell H Hannah Bonbrest A Amelie Essmann Z Zelai M. Garçon-Poca S Shinbe Choi M Michael R. Noya F Feonil Limiac A Ali Arce G Grant C. Glatfelter M Margaret Robinson L Li Chen A Angelina A. Mullarkey D Dain R. Brademan G Garrett Enten W William Dunne C César Quiroz I Ingrid Schoenborn C Chae Bin Lee R Rana Rais D Daniel P. Holt R Robert F. Dannals L Lei Shi (School of Health Management Guangzhou Medical University Guangzhou China) R Ruth Hüttenhain S Sergi Ferré E Eugene Kiyatkin J Jordi Bonaventura Y Yavin Shaham V Venetia Zachariou M Michael H. Baumann G Georgios Skiniotis K Kenner C. Rice M Michael Michaelides

Abstract

Abstract Developing safe and effective pain medications is an ongoing challenge for human health. Agonists for the µ-opioid receptor (MOR) are essential pain medications, but their high intrinsic efficacy also induces adverse side effects, including respiratory depression, constipation, tolerance, dependence, withdrawal and addiction 1–7 . Strategies to limit adverse effects traditionally include developing MOR agonists that have low intrinsic efficacy or that preferentially activate G-protein signalling over β-arrestin signalling 8 . Here we identify a novel MOR agonist with supramaximal intrinsic efficacy and a unique pharmacological profile that produced effective analgesia in rodents with minimal adverse effects. N -desethyl-fluornitrazene (DFNZ) was derived from a class of synthetic benzimidazole opioids called nitazenes. DFNZ has impaired brain penetrance, a unique spatiotemporal MOR cellular signalling profile, and diminished efficacy at the MOR–galanin 1 receptor (GAL1) heteromer. DFNZ does not induce respiratory depression, tolerance or MOR downregulation after repeated exposure. Compared with other MOR agonists, DFNZ has limited effects on dopamine neurotransmission in nucleus accumbens and weaker reinforcing effects in the drug self-administration procedure. These results provide novel insights about MOR and nitazene pharmacology, have important implications for pain and addiction treatment, and challenge the prevailing dogma that high-efficacy MOR agonists cannot constitute safe and effective therapeutic agents.

Article Details

Journal Nature
Volume / Issue Vol. 652, Issue 8112
Published April 30, 2026
Pages 1393-1404
ISSN 0028-0836
Publisher Nature Portfolio

Journal Info

Nature

Nature Portfolio

ISSN: 0028-0836 Health Sciences

Authors (44)

J

Juan L. Gomez

E

Emilya N. Ventriglia

Z

Zachary J. Frangos

A

Agnieszka Sulima

M

Michael J. Robertson

M

Michael D. Sacco

R

Reece C. Budinich

I

Ilinca M. Giosan

T

Tongzhen Xie

O

Oscar Solis

A

Anna E. Tischer

J

Jennifer M. Bossert

K

Kiera E. Caldwell

H

Hannah Bonbrest

A

Amelie Essmann

Z

Zelai M. Garçon-Poca

S

Shinbe Choi

M

Michael R. Noya

F

Feonil Limiac

A

Ali Arce

G

Grant C. Glatfelter

M

Margaret Robinson

L

Li Chen

A

Angelina A. Mullarkey

D

Dain R. Brademan

G

Garrett Enten

W

William Dunne

C

César Quiroz

I

Ingrid Schoenborn

C

Chae Bin Lee

R

Rana Rais

D

Daniel P. Holt

R

Robert F. Dannals

L

Lei Shi

School of Health Management Guangzhou Medical University Guangzhou China

R

Ruth Hüttenhain

S

Sergi Ferré

E

Eugene Kiyatkin

J

Jordi Bonaventura

Y

Yavin Shaham

V

Venetia Zachariou

M

Michael H. Baumann

G

Georgios Skiniotis

K

Kenner C. Rice

M

Michael Michaelides