A human-specific long noncoding RNA regulator of antigen-presenting cell viability and antimicrobial defense

A Alexander J. Westermann (Department of Microbiology, Biocenter, University of Würzburg) A Alexandra Schock (Institute for Molecular Infection Biology, Faculty of Medicine, University of Würzburg) D Diyaa Al Din Ashour (Department of Internal Medicine I, University Hospital Würzburg) S Sarah Wende (Institute for Lung Research, Faculty of Medicine, Philipps University Marburg) C Chrysanthi Skevaki (Institute of Clinical Chemistry and Laboratory Medicine, and Research Center for Immunotherapy, University Medical Center, Johannes Gutenberg-University Mainz) E Elisabeth Mack (Department of Hematology, Oncology and Immunology, Philipps University Marburg, University Hospital Giessen and Marburg) B Bernd Schmeck (Institute for Lung Research, Faculty of Medicine, Philipps University Marburg) U Uwe Linne (Mass spectrometry facility of the Department of Chemistry, Philipps University) T Thomas Herrmann (Institute for Virology and Immunobiology, Faculty of Medicine, Julius Maximilians Universität Würzburg) N Nikolaos Antonakos (4th Department of Internal Medicine, National and Kapodistrian University of Athens) H Helena Florou (Hellenic Institute for the Study of Sepsis) E Evangelos J. Giamarellos-Bourboulis (4th Department of Internal Medicine, National and Kapodistrian University of Athens) S Sebastian Weis (Institute for Infectious Disease and Infection Control, Jena University Hospital, Friedrich Schiller University) J Jörg Vogel L Leon N. Schulte (Institute for Lung Research, Faculty of Medicine, Philipps University Marburg)

Abstract

Macrophages are essential for both, to clear pathogens and preserve tissue homeostasis, yet the molecular regulators of this equilibrium remain incompletely defined. Here, we identify SAILR (survival associated immune-regulatory RNA), a primate-specific long noncoding RNA (lncRNA), as a critical modulator of macrophage viability under infection conditions. SAILR is induced during monocyte-to-macrophage differentiation, but rapidly downregulated upon bacterial challenge in a nuclear factor kappa B (NF-κB) dependent manner. In both naïve and immune-activated macrophages, SAILR dampens the expression of adhesion, phagocytosis, and invasion factors, which include SIGLEC1 and MMP7. During infection with Salmonella Typhimurium, depletion of SAILR sensitizes macrophages to apoptosis, resulting in loss of intracellular replication niches and reduced bacterial recovery. Conversely, enforced SAILR expression promotes macrophage survival and increases intracellular pathogen burden. Mechanistically, SAILR interacts with the antiapoptotic adaptor protein 14-3-3β to support macrophage survival. Notably, downregulation of SAILR is mirrored in circulating immune cells from patients with severe COVID-19 and sepsis. Together, our findings position SAILR as a central regulator in linking macrophage survival to host–pathogen interaction and disease pathophysiology.

Article Details

Volume / Issue Vol. 123, Issue 29
Published July 21, 2026
ISSN 0027-8424
Publisher National Academy of Sciences

Authors (15)

A

Alexander J. Westermann

Department of Microbiology, Biocenter, University of Würzburg

A

Alexandra Schock

Institute for Molecular Infection Biology, Faculty of Medicine, University of Würzburg

D

Diyaa Al Din Ashour

Department of Internal Medicine I, University Hospital Würzburg

S

Sarah Wende

Institute for Lung Research, Faculty of Medicine, Philipps University Marburg

C

Chrysanthi Skevaki

Institute of Clinical Chemistry and Laboratory Medicine, and Research Center for Immunotherapy, University Medical Center, Johannes Gutenberg-University Mainz

E

Elisabeth Mack

Department of Hematology, Oncology and Immunology, Philipps University Marburg, University Hospital Giessen and Marburg

B

Bernd Schmeck

Institute for Lung Research, Faculty of Medicine, Philipps University Marburg

U

Uwe Linne

Mass spectrometry facility of the Department of Chemistry, Philipps University

T

Thomas Herrmann

Institute for Virology and Immunobiology, Faculty of Medicine, Julius Maximilians Universität Würzburg

N

Nikolaos Antonakos

4th Department of Internal Medicine, National and Kapodistrian University of Athens

H

Helena Florou

Hellenic Institute for the Study of Sepsis

E

Evangelos J. Giamarellos-Bourboulis

4th Department of Internal Medicine, National and Kapodistrian University of Athens

S

Sebastian Weis

Institute for Infectious Disease and Infection Control, Jena University Hospital, Friedrich Schiller University

J

Jörg Vogel

L

Leon N. Schulte

Institute for Lung Research, Faculty of Medicine, Philipps University Marburg